Significant Involvement of CCL2 (MCP-1) in Inflammatory Disorders of the Lung
β Scribed by C. EDWARD ROSE JR.; SUNG-SANG J. SUNG; SHU MAN FU
- Book ID
- 110058675
- Publisher
- Informa plc
- Year
- 2010
- Tongue
- English
- Weight
- 119 KB
- Volume
- 10
- Category
- Article
- ISSN
- 1073-9688
No coin nor oath required. For personal study only.
π SIMILAR VOLUMES
## Abstract The loss of Eβcadherin expression and the translocation of Ξ²βcatenin to the nucleus are frequently associated with the metastatic conversion of epithelial cells. In the nucleus, Ξ²βcatenin binds to the TCF/LEFβ1 (Tβcell factor/ lymphoid enhancer factor) transcription factor family result
## Abstract Neural progenitor cells (NPCs) have been investigated as potential vehicles for brain tumor therapy because they have been shown to migrate toward central nervous system gliomas and can be genetically engineered to deliver cytotoxic agents to tumors. The mechanisms that regulate migrati
## Abstract Macrophages are critically involved in the pathogenesis of genetically caused demyelination, as it occurs in inherited demyelinating neuropathies. On the basis of the observation that upregulation of the Schwann cellβderived chemokine MCPβ1 (CCL2) is a pathologically relevant mechanism