𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Signal transduction and transcription factors in rheumatic disease

✍ Scribed by Gary S. Firestein; Anthony M. Manning


Publisher
John Wiley and Sons
Year
1999
Tongue
English
Weight
279 KB
Volume
42
Category
Article
ISSN
0004-3591

No coin nor oath required. For personal study only.


πŸ“œ SIMILAR VOLUMES


Nerve growth factor signal transduction
✍ H.H. Althaus; R. Hempel; S. KlΓΆppner; J. Engel; T. Schmidt-Schultz; L. Kruska; R πŸ“‚ Article πŸ“… 1997 πŸ› John Wiley and Sons 🌐 English βš– 350 KB πŸ‘ 1 views

It has previously been shown that nerve growth factor (NGF) is of functional significance for mature pig oligodendrocytes (OLs) in culture. The present data give evidence for the expression of TrkA, the so-called high-affinity NGF receptor, and of p75NTR, the socalled low-affinity NGF receptor. TrkA

Neurotrophin signal transduction in medu
✍ Thomas T. Chou; John Q. Trojanowski; Virginia M.-Y. Lee πŸ“‚ Article πŸ“… 1997 πŸ› John Wiley and Sons 🌐 English βš– 104 KB πŸ‘ 1 views

The members of the neurotrophin family play key biological roles in the development of the nervous system. Based on studies initially in cell lines (e.g., the rat pheochromocytoma PC12 cells), neurotrophins have been found to be important mediators of proliferation, differentiation, and survival in

Regulation ofLDH-A gene expression by tr
✍ Jungmann, Richard A.; Huang, Delai; Tian, Di πŸ“‚ Article πŸ“… 1998 πŸ› John Wiley and Sons 🌐 English βš– 218 KB

The lactate dehydrogenase-A (LDH-A) gene, whose product plays a pivotal role in normal anaerobic glycolysis and is frequently increased in human cancers, is highly regulated at the transcriptional and posttranscriptional levels. Our laboratory has carried out extensive studies concerning the regulat

TrkA signal transduction pathways in neu
✍ Eggert, Angelika ;Ikegaki, Naohiko ;Liu, Xing-ge ;Chou, Tom T. ;Brodeur, Garrett πŸ“‚ Article πŸ“… 2001 πŸ› John Wiley and Sons 🌐 English βš– 162 KB πŸ‘ 1 views

Background. Favorable neuroblastomas frequently express high levels of the TrkA receptor, and these tumors have a propensity to either differentiate or regress, but the mechanisms responsible for these two fates are unclear. Procedure. To study TrkA signal transduction in neuroblastoma (nb), we stab