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Sepsis inhibits recycling and glutamate-stimulated export of ascorbate by astrocytes

✍ Scribed by John X. Wilson; Magdalena Dragan


Book ID
113629637
Publisher
Elsevier Science
Year
2005
Tongue
English
Weight
295 KB
Volume
39
Category
Article
ISSN
0891-5849

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## Abstract Activated glial cells have been implicated in the neuropathogenesis of many infectious and inflammatory diseases of the brain. A number of inflammatory mediators have been proposed to play a role in glial cell‐related brain damage; e.g., free radicals such as nitric oxide (NO), cytokine