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Sensitivity of excision repair in normal human, xeroderma pigmentosum variant and Cockayne's syndrome fibroblasts to inhibition by cytosine arabinoside

✍ Scribed by James E. Cleaver


Publisher
John Wiley and Sons
Year
1981
Tongue
English
Weight
902 KB
Volume
108
Category
Article
ISSN
0021-9541

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✦ Synopsis


Inhibition of the gap-filling, polymerizing step of excision repair by 1-0-D-arabinofuranosylcytosine (ara-C) after irradiation with ultraviolet light in human diploid fibroblasts resulted in the formation of persistent DNA strand breaks in G,, G,, and plateau phase cells, but not in S phase cells. Addition of hydroxyurea to ara-C resulted in partial inhibition of repair in S phase cells.