𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Screening for self-renewal factors by a combination of mRNA and CGH microarray in human embryonic stem cells

✍ Scribed by Lei Xiao; Lixiazi He; Saul J. Sharkis

Publisher
SP Higher Education Press
Year
2010
Tongue
English
Weight
187 KB
Volume
5
Category
Article
ISSN
1674-7984

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Self-renewal of human embryonic stem cel
Self-renewal of human embryonic stem cells is supported by a shortened G1 cell cycle phase
✍ Klaus A. Becker; Prachi N. Ghule; Jaclyn A. Therrien; Jane B. Lian; Janet L. Ste 📂 Article 📅 2006 🏛 John Wiley and Sons 🌐 English ⚖ 585 KB

## Abstract Competency for self‐renewal of human embryonic stem (ES) cells is linked to pluripotency. However, there is a critical paucity of fundamental parameters of human ES cell division. In this study we show that human ES cells (H1 and H9; NIH‐designated WA01 and WA09) rapidly proliferate due

Cyclin D2 and the CDK substrate p220NPAT
Cyclin D2 and the CDK substrate p220NPAT are required for self-renewal of human embryonic stem cells
✍ Klaus A. Becker; Prachi N. Ghule; Jane B. Lian; Janet L. Stein; Andre J. van Wij 📂 Article 📅 2010 🏛 John Wiley and Sons 🌐 English ⚖ 476 KB

## Abstract Self‐renewal of pluripotent human embryonic stem (hES) cells utilizes an abbreviated cell cycle that bypasses E2F/pRB‐dependent growth control. We investigated whether self‐renewal is alternatively regulated by cyclin/CDK phosphorylation of the p220^NPAT^/HiNF‐P complex to activate __hi

Self-renewal and pluripotency is maintai
Self-renewal and pluripotency is maintained in human embryonic stem cells by co-culture with human fetal liver stromal cells expressing hypoxia inducible factor 1α
✍ Lei Ji; Yu-xiao Liu; Chao Yang; Wen Yue; Shuang-shuang Shi; Ci-xian Bai; Jia-fei 📂 Article 📅 2009 🏛 John Wiley and Sons 🌐 English ⚖ 1016 KB

## Abstract Human embryonic stem (hES) cells are typically maintained on mouse embryonic fibroblast (MEF) feeders or with MEF‐conditioned medium. However, these xenosupport systems greatly limit the therapeutic applications of hES cells because of the risk of cross‐transfer of animal pathogens. The