We previously demonstrated in the mouse oocyte that in vivo postovulatory aging significantly suppresses activity of the endoplasmic reticulum (ER) Ca 2ϩ -ATPase (Igarashi et al. 1997. Mol Reprod Dev 48:383-390). We undertook the present study to further examine the effects of oocyte aging on Ca 2ϩ
Role of 1,4,5-inositol triphosphate-induced Ca 2+ release in pollen tube orientation
✍ Scribed by Malhó, Rui
- Publisher
- Springer
- Year
- 1998
- Tongue
- English
- Weight
- 129 KB
- Volume
- 11
- Category
- Article
- ISSN
- 0934-0882
No coin nor oath required. For personal study only.
📜 SIMILAR VOLUMES
We have studied arginine vasopressin (AVP)-, thapsigarginand inositol 1,4,5trisphosphate (InsPJ-mediated CaL+ release in renal epithelial LLC-PK, cells. AVP-induced changes in the intracellular free calcium concentration ([Ca"],) were studied in indo-1 loaded single cells by confocal laser cytometry
## Abstract In non‐excitable cells, the inositol 1,4,5‐trisphosphate receptor (IP~3~R), a ligand‐gated Ca^2+^ channel, plays an important role in the control of intracellular Ca^2+^. There are three subtypes of IP~3~R that are differentially distributed among cell types. AR4‐2J cells express almost
## Abstract Studies with in‐vitro‐cultured neurons treated with amyloid‐β (Aβ) peptides demonstrated neuronal loss by apoptosis that is due, at least in part, to the perturbation of intracellular Ca^2+^ homeostasis. In addition, it was shown that an endoplasmic reticulum (ER)‐specific apoptotic pat
## Abstract The inositol 1,4,5‐trisphosphate receptor (IP~3~R), a ligand‐gated Ca^2+^ channel, is the main regulator of intracellular Ca^2+^ mobilization in non‐excitable cells. An emerging body of evidence suggests that specific regulatory control of the Ca^2+^ signaling pathway is modulated by th