๐”– Bobbio Scriptorium
โœฆ   LIBER   โœฆ

Rituximab sensitizes a Burkitt lymphoma cell line to cell killing by X-irradiation

โœ Scribed by Min Fengling; Liu Fenju; Wen Wanxin; Zhai Lijia; Tong Jiandong; Wang Zu; Yuan Xin; Gao Qingxiang


Publisher
Springer-Verlag
Year
2009
Tongue
English
Weight
427 KB
Volume
48
Category
Article
ISSN
0301-634X

No coin nor oath required. For personal study only.


๐Ÿ“œ SIMILAR VOLUMES


Resistance to etoposide-induced apoptosi
โœ E. G. Zhao; Q. Song; S. Cross; I. Misko; S. P. Lees-Miller; M. F. Lavin ๐Ÿ“‚ Article ๐Ÿ“… 1998 ๐Ÿ› John Wiley and Sons ๐ŸŒ French โš– 198 KB ๐Ÿ‘ 2 views

Burkitt's lymphoma cells that vary in their phenotypic characteristics show significantly different degrees of susceptibility to radiation-induced apoptosis. Propensity to undergo apoptosis is reflected in the degradation of substrates such as DNA-dependent protein kinase but the status of bcl-2, c-

Sensitivity to ebv superinfection and IU
โœ Okot Nyormoi; George Klein; Alice Adams; Laszlo Dombos ๐Ÿ“‚ Article ๐Ÿ“… 1973 ๐Ÿ› John Wiley and Sons ๐ŸŒ French โš– 1020 KB

## Abstract The sensitivity of Burkitt lymphomaโ€derived cell lines to Epsteinโ€Barr virus (EBV) superinfection and the inducibility of the latent EBV genome in these lines were studied by somatic cell hybridization. Two nonโ€EBV producer lines; AG R3, an adherent, 8โ€azaguanineโ€resistant variant of th

Irradiated fibroblasts protect burkitt l
โœ Martin H. Falk; Georg W. Bornkamm; Lothar Hรผltner; Anne Milner; Christopher D. G ๐Ÿ“‚ Article ๐Ÿ“… 1993 ๐Ÿ› John Wiley and Sons ๐ŸŒ French โš– 893 KB

When cells of fresh Burkitt lymphoma (BL) biopsies are explanted into tissue culture, their survival and growth are greatly dependent on the presence of a feeder layer of irradiated fibroblasts. To investigate the nature of this feeder dependence, we characterized the growth requirements of a panel

Wild-type p53-induced apoptosis in a Bur
โœ Olle Sangfelt; Stefan Einhorn; Ann Charlotte Bjรถrklund; Klas G. Wiman; Ismail Ok ๐Ÿ“‚ Article ๐Ÿ“… 1996 ๐Ÿ› John Wiley and Sons ๐ŸŒ French โš– 623 KB

The tumor suppressor p53 plays a central role in negative growth control, including growth arrest and apoptosis. Interferons (IFNs) are capable of modulating a variety of cellular responses, including apoptosis. In this study, we have evaluated the influence of y-and a-interferon (IFN) on wild-type