𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Reversibility and recurrence of IGF-IR-induced mammary tumors

✍ Scribed by Jones, R A; Campbell, C I; Wood, G A; Petrik, J J; Moorehead, R A

Book ID
109972291
Publisher
Nature Publishing Group
Year
2009
Tongue
English
Weight
837 KB
Volume
28
Category
Article
ISSN
0950-9232

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Absence of Ras mutations in rat DMBA-ind
Absence of Ras mutations in rat DMBA-induced mammary tumors
✍ Emma Samuelson; Johanna Nilsson; Anna Walentinsson; Claude Szpirer; Afrouz Behbo 📂 Article 📅 2009 🏛 John Wiley and Sons 🌐 English ⚖ 137 KB

## Abstract Animal cancer models reduce genetic background heterogeneity and thus, may facilitate identification and analysis of specific genetic aberrations in tumor cells. Rat and human mammary glands have high similarity in physiology and show comparable hormone responsiveness. Thus, spontaneous

Quantification of mouse mammary tumor vi
Quantification of mouse mammary tumor virus structural proteins in hormone-induced mammary tumors of low mammary tumor mouse strains
✍ R. Nüsse; R. Michalides; L. M. Boot; G. Röpcke 📂 Article 📅 1980 🏛 John Wiley and Sons 🌐 French ⚖ 663 KB

## Abstract The expression of the mouse mammary tumor virus (MMTV) in hormone‐induced mammary tumors was investigated by means of a radioimmunoassay for two major MMTV proteins, gp52 and p27. MMTV proteins were isolated on lectin affinity‐ and ion‐exchange chromatography columns. The purified viral

Characterization of medroxyprogesterone
Characterization of medroxyprogesterone and DMBA-induced multilineage mammary tumors by gene expression profiling
✍ Yuzhi Yin; Renkui Bai; Robert G. Russell; Marcy E. Beildeck; Zhihui Xie; Levy Ko 📂 Article 📅 2005 🏛 John Wiley and Sons 🌐 English ⚖ 505 KB

## Abstract Mouse mammary tumors arising during medroxyprogesterone‐DMBA‐mediated mammary carcinogenesis comprised three distinct phenotypes: adenocarcinoma, squamous cell carcinoma, and myoepithelial carcinoma. The molecular signature for each of the three tumor subsets was characterized by gene m