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Retrovirus-mediated WASP gene transfer corrects defective actin polymerization in B cell lines from Wiskott–Aldrich syndrome patients carrying ‘null’ mutations

✍ Scribed by Candotti, F; Facchetti, F; Blanzuoli, L; Stewart, D M; Nelson, D L; Blaese, R M

Book ID
110038435
Publisher
Nature Publishing Group
Year
1999
Tongue
English
Weight
357 KB
Volume
6
Category
Article
ISSN
0969-7128

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The Wiskott-Aldrich syndrome (WAS) is a rare X-linked recessive disorder characterized by eczema, thrombocytopenia, and immunodeficiency. An allelic variant of the disease is characterized by isolated thrombocytopenia (XLT). The gene responsible for WAS/XLT (WASP) encodes for a 502 amino acid protei