## Abstract The long QT syndrome gene human ether‐a‐go‐go related gene (__HERG__) encodes a K^+^ channel critical to cardiac repolarization. It peculiarly overexpresses in cancer cells of different histogenesis and promotes tumorigenesis. To decipher the molecular mechanisms for __HERG__ overexpres
Retraction: Overexpression HERG K+ channel gene mediates cell-growth signals on activation of oncoproteins SP1 and NF-κB and inactivation of tumor suppressor Nkx3.1
✍ Scribed by Huixian Lin; Jiening Xiao; Xiaobin Luo; Huizhen Wang; Huanhuan Gao; Baofeng Yang; Zhiguo Wang
- Publisher
- John Wiley and Sons
- Year
- 2011
- Tongue
- English
- Weight
- 39 KB
- Volume
- 227
- Category
- Article
- ISSN
- 0021-9541
No coin nor oath required. For personal study only.
✦ Synopsis
The retraction has been made due to the duplicated uses of previously published western blot bands, which violates the editorial policy of Journal of Cellular Physiology. The corresponding author, Zhiguo Wang, takes full responsibility and apologizes to the editors and readership of Journal of Cellular Physiology for any negative impact this may have on the journal.
📜 SIMILAR VOLUMES
## Abstract We previously reported that expression of angiogenesis factors interleukin‐8 (IL‐8) and vascular endothelial growth factor (VEGF) is promoted by coactivation of transcription factors nuclear factor‐kappaB (NF‐κB) and activator protein‐1 (AP‐1) by interleukin‐1α in human head and neck sq