✦ LIBER ✦

Restoration of the growth suppression function of mutant p53 by a synthetic peptide derived from the p53 C-terminal domain

✍ Scribed by Selivanova, Galina; Iotsova, Violetta; Okan, Ismail; Fritsche, Michael; Ström, Marika; Groner, Bernd; Grafström, Roland C.; Wiman, Klas G.

Book ID: 109929023
Publisher: Nature Publishing Group
Year: 1997
Tongue: English
Weight: 894 KB
Volume: 3
Category: Article
ISSN: 1078-8956
DOI: 10.1038/nm0697-632

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

ΔN-p53, a natural isoform of p53 la

ΔN-p53, a natural isoform of p53 lacking the first transactivation domain, counteracts growth suppression by wild-type p53

✍ Courtois, Stéphanie; Verhaegh, Gerald; North, Sophie; Luciani, Maria-Gloria; Las 📂 Article 📅 2002 🏛 Nature Publishing Group 🌐 English ⚖ 217 KB

Functional mimicry of the acetylated C-t

Functional mimicry of the acetylated C-terminal tail of p53 by a SUMO-1 acetylated domain, SAD

✍ Amrita Cheema; Chad D. Knights; Mahadev Rao; Jason Catania; Ricardo Perez; Brigi 📂 Article 📅 2010 🏛 John Wiley and Sons 🌐 English ⚖ 626 KB

## Abstract The ubiquitin‐like molecule, SUMO‐1, a small protein essential for a variety of biological processes, is covalently conjugated to many intracellular proteins, especially to regulatory components of the transcriptional machinery, such as histones and transcription factors. Sumoylation pr

Establishment of ponasterone A-inducible

Establishment of ponasterone A-inducible the wild-type p53 protein-expressing clones from HSC-1 cells, cell growth suppression by p53 expression and the suppression mechanism

✍ Makoto Hori; Keiji Suzuki; Masako U. Udono; Motohiro Yamauchi; Mariko Mine; Masa 📂 Article 📅 2008 🏛 Springer-Verlag 🌐 English ⚖ 854 KB

Tumor-derived p53 mutant C174Y is a gain

Tumor-derived p53 mutant C174Y is a gain-of-function mutant which activates the fos promoter and enhances colony formation

✍ Ute Preuss; Rene Kreutzfeld; Karl-Heinz Scheidtmann 📂 Article 📅 2000 🏛 John Wiley and Sons 🌐 French ⚖ 268 KB 👁 1 views

`Gain of function' phenotype of tumor-de

`Gain of function' phenotype of tumor-derived mutant p53 requires the oligomerization/nonsequence-specific nucleic acid-binding domain

✍ Lányi, Árpád; Deb, Debabrita; Seymour, Robert C; Ludes-Meyers, John H; Subler, M 📂 Article 📅 1998 🏛 Nature Publishing Group 🌐 English ⚖ 436 KB

Preferential induction of necrosis in hu

Preferential induction of necrosis in human breast cancer cells by a p53 peptide derived from the MDM2 binding site

✍ Do, Tamara N; Rosal, Ramon V; Drew, Lisa; Raffo, Anthony J; Michl, Josef; Pincus 📂 Article 📅 2003 🏛 Nature Publishing Group 🌐 English ⚖ 458 KB