✦ LIBER ✦

Requirement for ras proto-oncogene function during serum-stimulated growth of NIH 3T3 cells

✍ Scribed by Mulcahy, Linda S.; Smith, Mark R.; Stacey, Dennis W.

Book ID: 109737821
Publisher: Nature Publishing Group
Year: 1985
Tongue: English
Weight: 464 KB
Volume: 313
Category: Article
ISSN: 0028-0836
DOI: 10.1038/313241a0

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Raf-1 protein kinase is required for gro

Raf-1 protein kinase is required for growth of induced NIH/3T3 cells

✍ Kolch, Walter; Heidecker, Gisela; Lloyd, Patricia; Rapp, Ulf R. 📂 Article 📅 1991 🏛 Nature Publishing Group 🌐 English ⚖ 417 KB

Induction of p202, a modulator of apopto

Induction of p202, a modulator of apoptosis, during oncogenic transformation of NIH 3T3 cells by activated H-Ras (Q61L) contributes to cell survival

✍ Hong Xin; Yanbiao Geng; Rocky Pramanik; Divaker Choubey 📂 Article 📅 2002 🏛 John Wiley and Sons 🌐 English ⚖ 307 KB

## Abstract Previous studies have revealed that p202 (52 kDa), an interferon (IFN) and differentiation‐inducible protein, negatively regulates cell proliferation and modulates cell survival. However, the role of p202 in transformed cells remains to be investigated. Here we report that constitutive

Recessive (mediator−) revertants from c-

Recessive (mediator−) revertants from c-h-ras oncogene-transformed NIH 3T3 cells: Tumorigenicity in nude mice and transient anchorage and serum independence of the recovered tumor cells in culture

✍ Toshiko Omata-Yamada; Hisafumi Yamada; Peter Lengyel 📂 Article 📅 1991 🏛 John Wiley and Sons 🌐 English ⚖ 945 KB

## Abstract We have reported earlier the isolation of two recessive, serum‐ and anchoragedependent revertants (R116 and R260) from a c‐H‐ras oncogene‐transformed NIH 3T3 line. In both revertants, the oncogene was fully expressed and fusion of either revertant with (untransformed) NIH 3T3 cells, or