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Repair of 8-MOP cross-link damage in excision-defective strains of Escherichia coli

โœ Scribed by B.A. Bridges

Book ID
113250862
Publisher
Elsevier Science
Year
1984
Tongue
English
Weight
62 KB
Volume
130
Category
Article
ISSN
0165-1161

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The mutation recL152 leads to a reduction of excision repair as measured by an increase in the time required to close uvrA uvrB dependent incision breaks, and by a reduction of host cell reactivation ability. Postreplication repair is also delayed when measured in a uvrB5 recL152 double mutant. Such