The effect of riboflavin or pyridoxine deficiency on phagocytosis was examined in rats to elucidate the mechanism of impaired wound healing in these deficiencies reported earlier. Phagocytic ingestion by peritoneal leukocytes was 60.4% and 68%, respectively, in riboflavin-or pyridoxine-deficient rat
Renal lesions in pyridoxin-deficient rats
โ Scribed by Agnew, L. R. C.
- Publisher
- Wiley (John Wiley & Sons)
- Year
- 1951
- Tongue
- English
- Weight
- 660 KB
- Volume
- 63
- Category
- Article
- ISSN
- 0368-3494
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๐ SIMILAR VOLUMES
Male and female rats were fed a commercial diet lacking pyridoxine. Using radiological and histological methods the growth of the long bones in the pyridoxine-deficient rats from 2 wk to 6 mth of age was compared with that in paired-fed and in paired-weight-gain control rats of the same age and sex
The effect of riboflavin and (or) pyridoxine deficiency and repletion on tissue iron content was studied in rats. The iron content in liver, spleen, and kidney and plasma iron concentration of riboflavin deficient (RD) rats was lower, but hematocrit was not. In pyridoxine deficient (PD) rats versus
The non-hydroxy fatty acids of the galactolipid fraction of brain lipids of pyridoxine-deficient rats have been analyzed. The results suggest an impairment in the pathway for the synthesis of very long chain fatty acids in the brain of pyridoxine-deficient rats.
The effects of a large number of neurotropic drugs have been attributed to changes in the metabolism of 5-hydroxytryptamine. The aromatic amino acid decarboxylase considered to decarboxylate both dihydroxyphenylalanine and 5-hydroxytryptophan requires pyridoxal phosphate as coenzyme. Thus, in pyrido