Vascular Endothelial Growth Factor (VEGF) has been typically considered to be an endothelial-specific growth factor. However, it was recently demonstrated that VEGF can interact with non endothelial cells. In this study, we tested whether vascular smooth muscles cells (VSMCs) can express VEGF recept
Regulation of vascular endothelial cells and vascular smooth muscle cells by multiple P2Y receptor subtypes
β Scribed by Michael R. Boarder; Pamela J. White; Jonathon R. Roberts; Tania E. Webb
- Publisher
- John Wiley and Sons
- Year
- 2001
- Tongue
- English
- Weight
- 101 KB
- Volume
- 53
- Category
- Article
- ISSN
- 0272-4391
- DOI
- 10.1002/ddr.1186
No coin nor oath required. For personal study only.
π SIMILAR VOLUMES
## Abstract Intensive research efforts have been placed on the development of nanospheres for targeted drug delivery for treating a variety of diseases, including coronary restenosis, cancer, and inflammatory reactions. Although most of these drugβbearing spheres are delivered via intravenous admin
Glucose transport in response to angiotensin II (AII) was assessed in cultured vascular smooth muscle (VSM) cells by measuring the uptake of [ 3 H]-2-deoxyglucose, a radiolabeled non-metabolizable glucose analog. Significant stimulation occurred by 2 hr of exposure with the maximum effect being obse
## Abstract We have shown previously that nitricβoxide (NO) can induce apoptosis of vascular smooth muscle cells (VSMCs) and that the NOβinduced apoptosis is accompanied by an increase in arachidonic acid release via cytoplasmic Ca^2+^βdependent phospholipase A~2~ (cPLA~2~). We have evidence that d
In this study, we investigated the effect of the extracellular matrix (ECM) secreted by vascular cells on proteoglycan (PG) synthesis by vascular smooth muscle cells in culture. PG synthesis of human aortic smooth muscle cells plated on plastic or the matrices derived from vascular endothelial cells
The role of nutrient supply in the replicative capacity and secretory phenotype of cultured human diploid cells is unclear. We examined the relationship between amino acid privation, the secretion of vascular endothelial growth factor (VEGF) and growth phenotype of vascular smooth muscle cells (VSMC