Amyloid-b peptide is elevated in the brains of patients with Alzheimer disease and is believed to be causative in the disease process. Amyloid-b reduces glutamatergic transmission and inhibits synaptic plasticity, although the underlying mechanisms are unknown. We found that application of amyloid-b
✦ LIBER ✦
Regulation of NMDA receptor trafficking by amyloid-β
✍ Scribed by Snyder, Eric M; Nong, Yi; Almeida, Claudia G; Paul, Surojit; Moran, Timothy; Choi, Eun Young; Nairn, Angus C; Salter, Michael W; Lombroso, Paul J; Gouras, Gunnar K
- Book ID
- 109940653
- Publisher
- Nature Publishing Group
- Year
- 2005
- Tongue
- English
- Weight
- 365 KB
- Volume
- 8
- Category
- Article
- ISSN
- 1097-6256
- DOI
- 10.1038/nn1503
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