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Recombinant human tumor necrosis factor mediates regression of a murine sarcoma in vivo via Lyt-2+cells

✍ Scribed by A. L. Asher; J. J. Mulé; S. A. Rosenberg


Book ID
104660390
Publisher
Springer-Verlag
Year
1989
Tongue
English
Weight
427 KB
Volume
28
Category
Article
ISSN
0340-7004

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✦ Synopsis


The role of an immune response in recombinant-human-tumor-necrosis-factor(rHTNF)-mediated regression of a weakly immunogenic, MCA-106 sarcoma in vivo was examined. C57BL/6 mice bearing established 10-day s.c. tumor were treated with single i.v. doses (8 p~g) of rHTNF, rHTNF administration resulted in marked hemorrhagic necrosis and subsequent regression of tumor in treated mice. Mice cured of MCA-106 sarcoma by rHTNF specifically rejected a subsequent challenge (5 x 105 cells) of the same tumor (P <0.01) but not of the antigenically distinct, syngeneic MCA-105 sarcoma. Tumor bearers were depleted in vivo of selective T-cell subsets by the systemic administration of specific monoclonal antibodies before rHTNF therapy, rHTNF-induced regression, but not hemorrhagic necrosis of the MCA-106 sarcoma was blocked in mice depleted of Lyt-2 + cells, but not of L3T4 + cells. The in vivo role of T-cell subsets in rHTNF-mediated tumor regression is discussed.