𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Reactive oxygen species in TNFα-induced signaling and cell death

✍ Scribed by Michael J. Morgan; Zheng-gang Liu


Publisher
Korean Society for Molecular and Cellular Biology
Year
2010
Tongue
English
Weight
428 KB
Volume
30
Category
Article
ISSN
1016-8478

No coin nor oath required. For personal study only.


📜 SIMILAR VOLUMES


Vitamin D enhances caspase-dependent and
✍ Gregory E. Weitsman; Amiram Ravid; Uri A. Liberman; Ruth Koren 📂 Article 📅 2003 🏛 John Wiley and Sons 🌐 French ⚖ 225 KB

## Abstract Calcitriol, the hormonal form of vitamin D, potentiates the activity of some common anticancer drugs and agents of the anticancer immune system, including tumor necrosis factor α (TNFα). TNFα‐induced cytotoxicity is due to both caspase‐dependent and ‐independent pathways. Cotreatment wi

Reactive oxygen species induced by shear
✍ Susmita Sahoo; K. Krishnamurthy Rao; G.K. Suraishkumar 📂 Article 📅 2006 🏛 John Wiley and Sons 🌐 English ⚖ 276 KB

## Abstract Exposure of __Bacillus subtilis__ to a shear rate of 1,482/s leads to a rapid loss of cell viability after 10 h of growth. Biochemical and molecular evidences provided below strongly suggest that cell death under high shear results from an apoptosis‐like process similar to that describe

Reactive oxygen species as signals that
✍ Tsanko S. Gechev; Frank Van Breusegem; Julie M. Stone; Iliya Denev; Christophe L 📂 Article 📅 2006 🏛 John Wiley and Sons 🌐 English ⚖ 240 KB

Reactive oxygen species (ROS) are known as toxic metabolic products in plants and other aerobic organisms. An elaborate and highly redundant plant ROS network, composed of antioxidant enzymes, antioxidants and ROS-producing enzymes, is responsible for maintaining ROS levels under tight control. This

Astrocytes prevent neuronal death induce
✍ Junya Tanaka; Kazuko Toku; Bo Zhang; Ken Ishihara; Masahiro Sakanaka; Nobuji Mae 📂 Article 📅 1999 🏛 John Wiley and Sons 🌐 English ⚖ 653 KB

Reactive oxygen and nitrogen species (RO/NS) such as nitric oxide (NO), hydroxyl radical (OH•), and superoxide anion (O 2 Ϫ ) are generated in a variety of neuropathological processes and damage neurons. In the present study, we investigated the neuroprotective effects of rat astrocytes against RO/N