BACKGROUND. p53 is the most highly mutated tumor suppressor gene in human cancers. Recently, p73, a first homologue of p53, was identified and considered to be an imprinted tumor suppressor gene. Thus, we analyzed the possible role of p73 in human prostate cancers. METHODS. We investigated the expre
Quantitative analysis of a panel of gene expression in prostate cancer—with emphasis onNPYexpression analysis
✍ Scribed by Ai-jun Liu; Bungo Furusato; Lakshmi Ravindranath; Yong-mei Chen; Vasanta Srikantan; David G. Mcleod; Gyorgy Petrovics; Shiv Srivastava
- Book ID
- 111841076
- Publisher
- SP Zhejiang University Press
- Year
- 2007
- Tongue
- English
- Weight
- 264 KB
- Volume
- 8
- Category
- Article
- ISSN
- 1673-1581
No coin nor oath required. For personal study only.
📜 SIMILAR VOLUMES
Rapid advances in positional cloning studies have identified most of the genes on the human Y chromosome, thereby providing resources for studying the expression of its genes in prostate cancer. Using a semiquantitative reverse transcription±polymerase chain reaction (RT±PCR) procedure, we had exami
BACKGROUND. Loss of heterozygosity (LOH) on chromosome arm 18q is common in sporadic prostate cancer and may be involved in cancer development through inactivation of tumor-suppressor genes (TSG). Recent identification, at 18q21.1, of MADR2/Smad2, a key component in transforming growth factor  (TGF