𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Quantification of tropomyosin by radioimmunoassay in developing hearts of cardiac mutant axolotls,Ambystoma mexicanum

✍ Scribed by Pamela B. Moore; Larry F. Lemanski


Publisher
Springer Netherlands
Year
1982
Tongue
English
Weight
436 KB
Volume
3
Category
Article
ISSN
0142-4319

No coin nor oath required. For personal study only.


πŸ“œ SIMILAR VOLUMES


Analysis of the endocardium and cardiac
✍ Larry F. Lemanski; Timothy P. Fitzharris πŸ“‚ Article πŸ“… 1989 πŸ› John Wiley and Sons 🌐 English βš– 790 KB

Recessive mutant gene c in axolotls results in a failure of the heart to function because of abnormal embryonic induction processes. The myocardium in this mutant lacks organized sarcomeric myofibrils. The present study was undertaken to determine if developmental abnormalities were evident in other

Immunofluorescent, immunogold, and elect
✍ Pei-Shen Shen; Larry F. Lemanski πŸ“‚ Article πŸ“… 1989 πŸ› John Wiley and Sons 🌐 English βš– 825 KB

Recessive mutant gene c for "cardiac nonfunction" in axolotls results in an absence of normal heart contractions in affected embryos due to a failure of myofibril formation. In the present study, the intermediate filament protein, desmin, is compared in developing normal and mutant hearts by means o

Development of pigmentation in the eyele
✍ Epp, Leonard G. πŸ“‚ Article πŸ“… 1972 πŸ› John Wiley and Sons 🌐 English βš– 907 KB

## Abstract The development of pigment pattern in the eyeless mutant of __Ambystoma mexicanum__ is described. Pigmentation, is much enhanced in this mutant through increased size and branching of melanophores. The eyeless mutant does not possess the ability to adapt to its background, nor does it b

Molecular and immunohistochemical analys
✍ C. Zhang; K.M. Pietras; G.F. Sferrazza; P. Jia; G. Athauda; E. Rueda-de-Leon; J. πŸ“‚ Article πŸ“… 2007 πŸ› John Wiley and Sons 🌐 English βš– 573 KB

## Abstract The Mexican axolotl, __Ambystoma mexicanum__, is an excellent animal model for studying heart development because it carries a naturally occurring recessive genetic mutation, designated gene c, for cardiac nonfunction. The double recessive mutants (__c__/__c__) fail to form organized my

Analysis of actin and tropomyosin in hea
✍ Christopher M. Starr; Jose G. Diaz; Larry F. Lemanski πŸ“‚ Article πŸ“… 1989 πŸ› John Wiley and Sons 🌐 English βš– 751 KB

When homozygous, recessive mutant gene c in Ambystoma mexicanum results in a failure of embryonic heart function. This failure is apparently due to abnormal inductive influences from the anterior endoderm resulting in an absence of normal sarcomeric myofibril formation. Biochemical and immunofluores