✦ LIBER ✦

Pseudoneoplastic proliferation of endocrine cells in pancreatic fibrosis

✍ Scribed by Dr. Sue A. Bartow; Kiyoshi Mukai; Juan Rosai

Publisher: John Wiley and Sons
Year: 1981
Tongue: English
Weight: 879 KB
Volume: 47
Category: Article
ISSN: 0008-543X
DOI: 10.1002/1097-0142(19810601)47:11<2627::aid-cncr2820471118>3.0.co;2-c

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

The patterns of extrainsular endocrine c

The patterns of extrainsular endocrine cells in pancreatic cancer

✍ Bruno M. Schmied; Alexis B. Ulrich; Helmut Friess; Markus W. Büchler; Parviz M. 📂 Article 📅 2000 🏛 John Wiley and Sons 🌐 English ⚖ 249 KB 👁 2 views

Abnormal glucose tolerance and frank diabetes mellitus develop in up to 80% of pancreatic cancer patients. Islets within these tumors show a decreased number of beta cells and increased number of alpha cells. The reduced number of beta cells could induce beta cell neogenesis in extrainsular tissue t

Proliferation of parenchymal epithelial

Proliferation of parenchymal epithelial cells enhanced in chronic pancreatitis

✍ Slater, S. D.; Williamson, R. C. N.; Foster, C. S. 📂 Article 📅 1998 🏛 John Wiley and Sons 🌐 English ⚖ 488 KB 👁 1 views

This study was performed to determine whether pancreatic parenchymal epithelial cells in human chronic pancreatitis tissues retain a biologically significant capability to proliferate and, if so, within which epithelial compartment proliferation occurs. The techniques of immediate per-operative in v

Gefitinib Inhibits the Proliferation of

Gefitinib Inhibits the Proliferation of Pancreatic Cancer Cells via Cell Cycle Arrest

✍ Xiaohua Zhou; Maqing Zheng; Fang Chen; Yunxia Zhu; Wei Yong; Haiyan Lin; Yujie S 📂 Article 📅 2009 🏛 Wiley (John Wiley & Sons) 🌐 English ⚖ 281 KB 👁 2 views

## Abstract High expression of the epidermal growth factor receptor (EGFR) has been implicated in the development of pancreatic cancer. Gefitinib is an orally active and selective EGFR‐TKI (EGFR‐tyrosine kinase inhibitor) that blocks signal transduction pathways responsible for the proliferation an

Genomic imbalances in the progression of

Genomic imbalances in the progression of endocrine pancreatic tumors

✍ Jianming Zhao; Holger Moch; Alexander F. Scheidweiler; Angela Baer; Alejandro A. 📂 Article 📅 2001 🏛 John Wiley and Sons 🌐 English ⚖ 200 KB

## Abstract Endocrine pancreatic tumors (EPTs) are neoplasms with malignant potential. To explore the molecular basis of metastatic progression in human EPTs, we analyzed 17 paired specimens of primary EPTs and their metastases and 28 nonmetastatic EPTs using comparative genomic hybridization (CGH)

Conversion of immortal liver progenitor

Conversion of immortal liver progenitor cells into pancreatic endocrine progenitor cells by persistent expression of Pdx-1

✍ Cai-Xia Jin; Wen-Lin Li; Fang Xu; Zhen H. Geng; Zhi-Ying He; Juan Su; Xin-Rong T 📂 Article 📅 2008 🏛 John Wiley and Sons 🌐 English ⚖ 425 KB

## Abstract The conversion of expandable liver progenitor cells into pancreatic beta cells would provide a renewable cell source for diabetes cell therapy. Previously, we reported the establishment of liver epithelial progenitor cells (LEPCs). In this work, LEPCs were modified into EGFP/Pdx‐1 LEPCs

Hyperglycemia and hyperinsulinemia have

Hyperglycemia and hyperinsulinemia have additive effects on activation and proliferation of pancreatic stellate cells: Possible explanation of islet-specific fibrosis in type 2 diabetes mellitus

✍ Oak-Kee Hong; Seung-Hwan Lee; Marie Rhee; Seung-Hyun Ko; Jae-Hyoung Cho; Yoon-He 📂 Article 📅 2007 🏛 John Wiley and Sons 🌐 English ⚖ 301 KB