## Abstract Nitric oxide (NO) can regulate chondrocyte activities. This study was aimed to evaluate the molecular mechanisms of NO donor sodium nitroprusside (SNP)‐induced insults to human chondrocytes. Exposure of human chondrocytes to SNP increased cellular NO levels but decreased cell viability
Protein kinase C-mediated tyrosine phosphorylation of paxillin and focal adhesion kinase requires cytoskeletal integrity and is uncoupled to mitogen-activated protein kinase activation in human hepatoma cells
✍ Scribed by Lan Chun Tu; Chen-Kung Chou; Hua-Chien Chen; Sheau-Farn Yeh
- Publisher
- BioMed Central
- Year
- 2001
- Tongue
- English
- Weight
- 947 KB
- Volume
- 8
- Category
- Article
- ISSN
- 1021-7770
No coin nor oath required. For personal study only.
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