Protein deficiency and muscle damage in carbon tetrachloride induced liver cirrhosis
✍ Scribed by A. López-Lirola; E. González-Reimers; R. Martı́n Olivera; F. Santolaria-Fernández; L. Galindo-Martı́n; P. Abreu-González; T. González-Hernández; F. Valladares-Parrilla
- Book ID
- 117450275
- Publisher
- Elsevier Science
- Year
- 2003
- Tongue
- English
- Weight
- 225 KB
- Volume
- 41
- Category
- Article
- ISSN
- 0278-6915
No coin nor oath required. For personal study only.
📜 SIMILAR VOLUMES
Previous studies have shown that a-tocopherol (vitamin E) pretreatment of experimental animals can protect against acute liver necrosis induced by carbon tetrachloride. In this study we investigated whether the increase of vitamin E liver content by dietary supplementation influences chronic liver d
Cirrhosis is the most common end stage of liver diseases, and there are no effective treatment methods. Here we evaluated the effect of endothelial progenitor cell (EPC) transplantation from rat bone marrow (BM) on the development of cirrhosis induced by carbon tetrachloride (CCl 4 ). Ex vivo genera
Inhibition of Kupffer cells could disrupt the sequence of events leading to organ injury by damping down the fibrogenic stimulus. To elucidate the role of Kupffer cells in liver fibrosis and cirrhosis, rats were treated with gadolinium chloride (GdCl(3)) and cirrhosis was induced by subchronic carbo