✦ LIBER ✦

Protection from olanzapine-induced metabolic toxicity in mice by acetaminophen and tetrahydroindenoindole

✍ Scribed by Shertzer, H G; Kendig, E L; Nasrallah, H A; Johansson, E; Genter, M B

Book ID: 109866556
Publisher: Nature Publishing Group
Year: 2010
Tongue: English
Weight: 614 KB
Volume: 34
Category: Article
ISSN: 1476-5497
DOI: 10.1038/ijo.2009.291

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Mitochondrial protection by the JNK inhi

Mitochondrial protection by the JNK inhibitor leflunomide rescues mice from acetaminophen-induced liver injury

✍ Calivarathan Latchoumycandane; Catherine W. Goh; Michie M.K. Ong; Urs A. Boelste 📂 Article 📅 2007 🏛 John Wiley and Sons 🌐 English ⚖ 842 KB

Acetaminophen (APAP) is a widely used analgesic and antipyretic drug that is safe at therapeutic doses but which can precipitate liver injury at high doses. We have previously found that the antirheumatic drug leflunomide is a potent inhibitor of APAP toxicity in cultured human hepatocytes, protecti

Attenuation of 2-methoxyethanol and meth

Attenuation of 2-methoxyethanol and methoxyacetic acid-induced digit malformations in mice by simple physiological compounds: Implications for the role of further metabolism of methoxyacetic acid in developmental toxicity

✍ Welsch, F. ;Sleet, R. B. ;Greene, J. A. 📂 Article 📅 1987 🏛 John Wiley and Sons 🌐 English ⚖ 884 KB

The ethylene glycol ether 2-methoxyethanol (ME) and its oxidation product methoxyacetic acid (MAA) are selective embryotoxins and equipotent as inducers of digit malformations when given by gavage to pregnant Cr1:CD-1 ICR BR mice on gestation day 11. Earlier observations showed that the teratogenic