𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Promotion of Hepatocarcinogenesis by Phenobarbital in c-myc/TGF-α Transgenic Mice

✍ Scribed by Sean Sanders; Snorri S. Thorgeirsson


Publisher
John Wiley and Sons
Year
2000
Tongue
English
Weight
258 KB
Volume
28
Category
Article
ISSN
0899-1987

No coin nor oath required. For personal study only.


📜 SIMILAR VOLUMES


Upregulation of endogenous p53 and induc
✍ V. S. Prasad; Richard E. LaFond; Ming Zhou; Karen A. Jacobsen; Dennis G. Osmond; 📂 Article 📅 1997 🏛 John Wiley and Sons 🌐 English ⚖ 483 KB

Eµ-myc transgenic mice carry a constitutively overexpressed c-myc oncogene and develop B-lineage lymphomas. Previous studies have shown that c-myc overexpression can lead to in vitro apoptosis. Here, we investigated the in vivo effects of altered c-myc expression on cell proliferation versus death i

Expression of c-myc in altered hepatic f
✍ Takeshi Deguchi; Henry C. Pitot 📂 Article 📅 1995 🏛 John Wiley and Sons 🌐 English ⚖ 847 KB

## Abstract Among the proto‐oncogenes examined by northern blot analysis, c‐__myc__, c‐Ha‐__ras__, c‐__fos__, and c‐__raf__‐1 have been reported to be activated in rat liver cell carcinomas. However, there are relatively few reports on proto‐oncogene expression in altered hepatic foci (a) early dur

Hepatitis C virus core protein induces s
✍ Naoki Tanaka; Kyoji Moriya; Kendo Kiyosawa; Kazuhiko Koike; Toshifumi Aoyama 📂 Article 📅 2007 🏛 John Wiley and Sons 🌐 French ⚖ 493 KB

## Abstract Persistent infection of hepatitis C virus (HCV) can lead to a high risk for hepatocellular carcinoma (HCC). HCV core protein plays important roles in HCV‐related hepatocarcinogenesis, because mice carrying the core protein exhibit multicentric HCCs without hepatic inflammation and fibro

Impairment of mammary lobular developmen
✍ Valeria Buggiano; Carolina Schere-Levy; Keiji Abe; Silvia Vanzulli; Isabel Piazz 📂 Article 📅 2001 🏛 John Wiley and Sons 🌐 French ⚖ 551 KB

It has previously been shown that transgenic female mice expressing TGF␤1 under control of regulatory elements of the whey-acidic protein (WAP) gene were unable to lactate. This was due to the increased apoptosis of the cells committed to the lobular-lactogenic phenotype. Our goal was to determine w