## Abstract ## BACKGROUND Antiapoptotis resulting from hyperactivation of the transcription factor NF‐κB has been described in several cancer types. It is triggered by the interaction of the tumor necrosis factor (TNF) with its receptors and recruitment of the intermediate factor TNF‐receptor asso
Pro-apoptotic action of PAR-4 involves inhibition of NF-κB activity and suppression of BCL-2 expression
✍ Scribed by Simonetta Camandola; Mark P. Mattson
- Publisher
- John Wiley and Sons
- Year
- 2000
- Tongue
- English
- Weight
- 126 KB
- Volume
- 61
- Category
- Article
- ISSN
- 0360-4012
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