๐”– Bobbio Scriptorium
โœฆ   LIBER   โœฆ

Primary human cervical carcinoma cells require human papillomavirus E6 and E7 expression for ongoing proliferation

โœ Scribed by Thomas G. Magaldi; Laura L. Almstead; Stefania Bellone; Edward G. Prevatt; Alessandro D. Santin; Daniel DiMaio

Book ID
113948379
Publisher
Elsevier Science
Year
2012
Tongue
English
Weight
990 KB
Volume
422
Category
Article
ISSN
0042-6822

No coin nor oath required. For personal study only.

๐Ÿ“œ SIMILAR VOLUMES

Radiation-enhanced expression of E6/E7 t
Radiation-enhanced expression of E6/E7 transforming oncogenes of human papillomavirus-16 in human cervical carcinoma
โœ Alessandro D. Santin; Paul L. Hermonat; Antonella Ravaggi; Maurizio Chiriva-Inte ๐Ÿ“‚ Article ๐Ÿ“… 1998 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 107 KB ๐Ÿ‘ 2 views

## BACKGROUND. Human papillomavirus (HPV) infection represents the most important risk factor for cervical carcinoma. Levels of expression of E6 and E7 transforming oncoproteins of high risk HPV genotypes (i.e., HPV-16 and HPV-18) have been linked specifically to the mitotic activity of cervical c

Fragile histidine triad transcription ab
Fragile histidine triad transcription abnormalities and human papillomavirus E6โ€“E7 mRNA expression in the development of cervical carcinoma
โœ Tomoya Segawa; Toshiyuki Sasagawa; Hiroshi Yamazaki; Jun Sakaike; Hiroshi Ishika ๐Ÿ“‚ Article ๐Ÿ“… 1999 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 627 KB

## BACKGROUND. Human papillomaviruses (HPV) are the most important etiologic factor for cervical carcinoma. However, additional cellular events may be necessary for malignant progression in the cervix. Recently, the fragile histidine triad (FHIT) gene, which is frequently lost in many cancers, was