𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Prevention of Glucocorticoid-Induced Apoptosis in Osteocytes and Osteoblasts by Calbindin-D28k

✍ Scribed by Yan Liu; Angela Porta; Xiaorong Peng; Kristen Gengaro; Earlene B Cunningham; Hong Li; Luis A Dominguez; Teresita Bellido; Sylvia Christakos


Book ID
111485004
Publisher
American Society for Bone and Mineral Research
Year
2003
Tongue
English
Weight
807 KB
Volume
19
Category
Article
ISSN
0884-0431

No coin nor oath required. For personal study only.


πŸ“œ SIMILAR VOLUMES


Transgenic disruption of glucocorticoid
✍ Frank Buttgereit; Hong Zhou; Robert Kalak; Timo Gaber; Cornelia M. Spies; DΓΆrte πŸ“‚ Article πŸ“… 2009 πŸ› John Wiley and Sons 🌐 English βš– 430 KB

## Abstract ## Objective Endogenous glucocorticoids (GCs) modulate numerous biologic systems involved in the initiation and maintenance of arthritis. Bone cells play a critical role in the progression of arthritis, and some of the effects of GCs on inflammation may be mediated via these cells. The

Apoptosis- and endoplasmic reticulum str
✍ Eui-Man Jung; Beum-Soo An; Kyung-Chul Choi; Eui-Bae Jeung πŸ“‚ Article πŸ“… 2011 πŸ› John Wiley and Sons 🌐 English βš– 614 KB

## Abstract Calcium (Ca^2+^) is an important regulator of apoptotic signaling. Calbindin‐D~9k~ (CaBP‐9k) and ‐D~28k~ (CaBP‐28k) have a high affinity for Ca^2+^ ions. Uterine calbindins appear to be involved in the regulation of myometrial activity by intracellular Ca^2+^. In addition, uterine calbi