๐”– Bobbio Scriptorium
โœฆ   LIBER   โœฆ

Prevalence of atopic eczema in the community: the Lothian atopic dermatitis study

โœ Scribed by R.M. HERD; M.J. TIDMAN; R.J. PRESCOTT; J.A.A. HUNTER


Publisher
John Wiley and Sons
Year
1996
Tongue
English
Weight
187 KB
Volume
135
Category
Article
ISSN
0007-0963

No coin nor oath required. For personal study only.


๐Ÿ“œ SIMILAR VOLUMES


The descriptive epidemiology of atopic d
โœ Adrian Mar; Robin Marks ๐Ÿ“‚ Article ๐Ÿ“… 1999 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 71 KB

Atopic dermatitis (AD) is a common condition in the community, particularly amongst children. Although comparison of prevalence data between surveys is made difficult by differences in methodology, the available data suggest that there has been a substantial rise in the prevalence of AD, and that so

Prevalence of atopic dermatitis, asthma,
โœ C.G. Mortz; J.M. Lauritsen; C. Bindslev-Jensen; K.E. Andersen ๐Ÿ“‚ Article ๐Ÿ“… 2001 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 357 KB

## Background: Atopic diseases are common in children and adolescents. however, epidemiological knowledge is sparse for hand eczema and allergic contact dermatitis in this age group. furthermore, no population-based studies have evaluated the prevalence of atopic diseases and hand and contact derma

Flucloxacillin in the treatment of atopi
โœ Ewing; Ashcroft; Gibbs; Jones; Connor; David ๐Ÿ“‚ Article ๐Ÿ“… 1998 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 479 KB

Although colonization of atopic dermatitis by Staphylococcus aureus is universal and bacterial infection is common, it is not known whether antibiotic therapy is helpful in eczematous children who do not have any signs suggestive of bacterial infection. Fifty children aged 1-16 years with atopic der

New concepts in the pathogenesis of atop
โœ Donald YM Leung; Neal Jain; Harvey L Leo ๐Ÿ“‚ Article ๐Ÿ“… 2003 ๐Ÿ› Elsevier Science ๐ŸŒ English โš– 114 KB

Atopic dermatitis is a chronic, relapsing, inflammatory skin disease that is characterized by pruritic, eczematoid skin lesions. The disease results from interactions between susceptibility genes, the host environment, skin barrier defects, susceptibility to infection and immunological factors. An i