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Preserving Neurocognitive Functions after Traumatic Brain Injury through Modulation of Acetylation in a Rodent Model

✍ Scribed by M. Poloujadoff; J. Khuman; B. Liu; J. Zhang; J. Park; E. Fukudome; M. deMoya; M. Whalen; H.B. Alam

Book ID
116705750
Publisher
Elsevier Science
Year
2010
Tongue
English
Weight
109 KB
Volume
158
Category
Article
ISSN
0022-4804

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## Abstract Hippocampal N‐methyl‐D‐aspartate (NMDA) receptor subunits, by virtue of their involvement in excitotoxic injury as well as memory association, may play an important role in the pathophysiologic mechanisms of traumatic brain injury (TBI). In this study, temporal changes in NMDA receptor