Presenilin mutations associated with Alzheimer disease cause defective intracellular trafficking of β-catenin,a component of the presenilin protein complex

✍ Scribed by St George–Hyslop, P.; Nishimura, M.; Yu, G.; Levesque, G.; Zhang, D.M.; Ruel, L.; Chen, F.; Milman, P.; Holmes, E.; Liang, Y.; Kawarai, T.; Jo, E.; Supala, A.; Rogaeva, E.; Xu, D -M.; Janus, C.; Levesque, L.; Bi, Q.; Duthie, M.; Rozmahel, R.; Mattila, K.; Lannfelt, L.; Westaway, D.; Mount, H.T.J.; Woodgett, J.; Fraser, P.E.

• Book ID: 109826703
• Publisher: Nature Publishing Group
• Year: 1999
• Tongue: English
• Weight: 311 KB
• Volume: 5
• Category: Article
• ISSN: 1078-8956
• DOI: 10.1038/5526