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Presenilin-1 but not amyloid precursor protein mutations present in mouse models of Alzheimer’s disease attenuate the response of cultured cells to γ-secretase modulators regardless of their potency and structure

✍ Scribed by Stefanie Hahn; Tanja Brüning; Julia Ness; Eva Czirr; Sandra Baches; Harrie Gijsen; Carsten Korth; Claus U. Pietrzik; Bruno Bulic; Sascha Weggen


Book ID
111184346
Publisher
John Wiley and Sons
Year
2010
Tongue
English
Weight
441 KB
Volume
116
Category
Article
ISSN
0022-3042

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