## Abstract Neonatal hypoxic‐ischemic (HI) white matter injury is a major contributor to chronic neurological dysfunction. Immature oligodendrocytes (OLGs) are highly vulnerable to HI injury. As little is known about in vivo OLG repair mechanisms in neonates, we studied whether new OLGs are generat
✦ LIBER ✦
Pontine axonal injury after brain trauma and nontraumatic hypoxic-ischemic brain damage
✍ Scribed by M. Oehmichen; C. Meißner; V. Schmidt; I. Pedal; H. G. König
- Publisher
- Springer-Verlag
- Year
- 1999
- Tongue
- German
- Weight
- 178 KB
- Volume
- 112
- Category
- Article
- ISSN
- 0937-9827
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Iron can contribute to hypoxic-ischemic brain damage by catalyzing the formation of free radicals. The immature brain has high iron levels and limited antioxidant defenses. The objective of this study was to describe the early alterations in nonheme iron histochemistry following a hypoxic-ischemic (