## Abstract ## Objective Neutrophils and tumor necrosis factor (TNF) play important roles in the pathogenesis of rheumatoid arthritis (RA). Modulation of TNF receptors (TNFRs) may contribute to the regulation of tissue damage, and __n__โ6 polyunsaturated fatty acids (PUFAs) such as arachidonic aci
โฆ LIBER โฆ
Polyunsaturated fatty acids stimulate superoxide formation in tumor cells: A mechanism for specific cytotoxicity and a model for tumor necrosis factor?
โ Scribed by D.A. Peterson; N. Mehta; J. Butterfield; M. Husak; M.M. Christopher; S. Jagarlapudi; J.W. Eaton
- Book ID
- 117058149
- Publisher
- Elsevier Science
- Year
- 1988
- Tongue
- English
- Weight
- 271 KB
- Volume
- 155
- Category
- Article
- ISSN
- 0006-291X
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## Abstract As previously shown, sera from tumorโbearing mice can induce specific antiserumโdependent lymphoid cellโmediated cytotoxicity (ADC) to syngeneic tumor cells in vitro. The ADC activity in such sera is now shown to be removed by adsorption of the sera to Sepharoseโlinked rabbit antiโmouse