Polyglandular Autoimmune Syndrome Type I

Progressive vascular calcification in au

Progressive vascular calcification in autoimmune polyglandular syndrome type I

✍ S. Ohga; Naoko Matsumoto; Hidetoshi Takada; Akihiko Nomura; Tetsuo Matsuda; Tosh 📂 Article 📅 2001 🏛 Springer-Verlag 🌐 English ⚖ 50 KB

Pancytopenia and Myelofibrosis in Autoim

Pancytopenia and Myelofibrosis in Autoimmune Polyglandular Syndrome Type I

✍ Braskett, M.; Garcia-Lloret, M.I.; Howenstine, S.; McCurdy, D.; McGhee, S. 📂 Article 📅 2007 🏛 Elsevier Science 🌐 English ⚖ 49 KB

Erythropoietin-deficient anemia associat

Erythropoietin-deficient anemia associated with autoimmune polyglandular syndrome type I

✍ Rebecca Toonkel; Michael Levine; Lawrence Gardner 📂 Article 📅 2004 🏛 John Wiley and Sons 🌐 English ⚖ 77 KB 👁 2 views

Reversible hyperkinesia in a patient wit

Reversible hyperkinesia in a patient with autoimmune polyglandular syndrome type I

✍ Baumert, T. ;Kleber, G. ;Schwarz, J. ;St�bler, A. ;Lamerz, R. ;Mann, K. 📂 Article 📅 1993 🏛 Springer-Verlag 🌐 English ⚖ 554 KB

Autoimmune polyglandular syndrome is characterized by a failure of multiple endocrine organs and the presence of circulating organ-specific autoantibodies targeted against the failing organs. Here we describe a patient with autoimmune polyglandular syndrome type I with the endocrine manifestations o

Autoantibodies in autoimmune polyglandul

Autoantibodies in autoimmune polyglandular syndrome type I patients react with major brain neurotransmitter systems

✍ Sergueï O. Fetissov; Sophie Bensing; Jan Mulder; Erwan Le Maitre; Anna-Lena Hult 📂 Article 📅 2009 🏛 John Wiley and Sons 🌐 English ⚖ 321 KB

## Abstract Patients with autoimmune polyglandular syndrome type I (APS1) often display high titers of autoantibodies (autoAbs) directed against aromatic L‐amino acid decarboxylase (AADC), tyrosine hydroxylase (TH), tryptophan hydroxylase (TPH), and glutamic acid decarboxylase (GAD). Neurological s

Autoantibodies in autoimmune polyglandul

Autoantibodies in autoimmune polyglandular syndrome type I patients react with major brain neurotransmitter systems

✍ Sergueï O. Fetissov; Sophie Bensing; Jan Mulder; Erwan Le Maitre; Anna-Lena Hult 📂 Article 📅 2009 🏛 John Wiley and Sons 🌐 English ⚖ 320 KB

## Abstract Patients with autoimmune polyglandular syndrome type I (APS1) often display high titers of autoantibodies (autoAbs) directed against aromatic L‐amino acid decarboxylase (AADC), tyrosine hydroxylase (TH), tryptophan hydroxylase (TPH), and glutamic acid decarboxylase (GAD). Neurological s