Phenotypic analyses of lymphokine-activated killer cells that release interferon γ and tumor necrosis factor α
✍ Scribed by Anita S. -F. Chong; Belen Ybarrondo; William J. Grimes; Evan M. Hersh; Philip Scuderi
- Publisher
- Springer-Verlag
- Year
- 1990
- Tongue
- English
- Weight
- 682 KB
- Volume
- 31
- Category
- Article
- ISSN
- 0340-7004
No coin nor oath required. For personal study only.
📜 SIMILAR VOLUMES
With the aim of developing an effective immunotherapy for malignant glioma, glioma cells were incubated with tumor necrosis factor-a (TNF-a) to increase their susceptibility to lysis by lymphokine-activated killer (LAK) cells. Treatment with exogenous TNF-a induced the expression of intercellular ad
While it is well known that interferon-␥ (IFN␥) and tumor necrosis factor-␣ (TNF␣) play a role in the regulation of thyroid growth and differentiated functions, the cellular and molecular mechanisms involved in mediating the effects of IFN␥ and TNF␣ on thyroid function are unknown. In the present st
Toxoplasrna gondii, an obligate intracellular parasite, is able to replicate in human brain cells. We recently showed that interferon (1FN)-y-activated cells from glioblastoma line 86HG39 were able to restrict Toxoplusma growth. The effector mechanism responsible for this toxoplasmostatic effect was