๐”– Bobbio Scriptorium
โœฆ   LIBER   โœฆ

Paget'S disease of the skin. A unifying concept of histogenesis

โœ Scribed by Mehrdad Nadji; Azorides R. Morales; Robert E. Girtanner; Jocelyne Ziegels-Weissman; Neal S. Penneys


Publisher
John Wiley and Sons
Year
1982
Tongue
English
Weight
367 KB
Volume
50
Category
Article
ISSN
0008-543X

No coin nor oath required. For personal study only.


๐Ÿ“œ SIMILAR VOLUMES


Scrape cytology of pemphigus vulgaris of
โœ Tadao K. Kobayashi; Masami Ueda; Toshihiro Nishino; Sachiko Kibe; Takashi Higash ๐Ÿ“‚ Article ๐Ÿ“… 1997 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 226 KB ๐Ÿ‘ 1 views

A case of histologically and immunocytochemically confirmed pemphigus vulgaris of the nipple examined by scrape cytology is reported. The scrape cytology showed isolated and somewhat loosely clustered cells with basophilic cytoplasm and vesicular hyperchromatic nuclei dysplaying prominent nucleoli,

The conservative management of Paget's d
โœ Lori J. Pierce; Bruce G. Haffty; Lawrence J. Solin; Beryl McCormick; Frank A. Vi ๐Ÿ“‚ Article ๐Ÿ“… 1997 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 100 KB ๐Ÿ‘ 1 views

## RESULTS. The median follow-up for surviving patients was 62 months. Three pa- ## Michigan. tients (10%) developed a recurrence in the breast as the only site of first failure,

A unifying hypothesis of Alzheimer's dis
โœ Kurt Heininger ๐Ÿ“‚ Article ๐Ÿ“… 2000 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 484 KB ๐Ÿ‘ 1 views

Normal ageing and Alzheimer's disease (AD) have many features in common and, in many respects, both conditions only differ by quantitative criteria. A variety of genetic, medical and environmental factors modulate the ageing-related processes leading the brain into the devastation of AD. In accordan

A unifying hypothesis of Alzheimer's dis
โœ Kurt Heininger ๐Ÿ“‚ Article ๐Ÿ“… 1999 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 472 KB ๐Ÿ‘ 1 views

In a series of ยฎve papers, evidence from neurobiology, endocrinology and immunology is integrated into a holistic, coherent hypothesis accommodating genetic, medical and environmental risk factors into a cascade of pathophysiological events that leads to the clinical manifestation of Alzheimer's dis