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p73 G4C14-to-A4T14 polymorphism and risk of human papillomavirus-associated squamous cell carcinoma of the oropharynx in never smokers and never drinkers

✍ Scribed by Xingming Chen; Erich M. Sturgis; Carol J. Etzel; Qingyi Wei; Guojun Li

Publisher
John Wiley and Sons
Year
2008
Tongue
English
Weight
109 KB
Volume
113
Category
Article
ISSN
0008-543X

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## Abstract ## BACKGROUND: The tumor protein p73 interacts with the human papillomavirus type 16 (HPV‐16) oncoproteins E6 and E7, and __p73__ variation may modify the interaction between p73 protein and HPV‐16 oncogenic proteins and contribute to cellular malignant transformation. ## METHODS: In

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## Abstract P73 plays an important role in modulating cell‐cycle control, inducing apoptosis, and inhibiting cell growth. A novel noncoding __p73__ G4C14‐to‐A4T14 exon 2 polymorphism was associated with risk of squamous cell carcinoma of the head and neck (SCCHN). We hypothesized that __p73__ G4C14