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p38 MAPK inhibition is critically involved in VEGFR-2-mediated endothelial cell survival

✍ Scribed by Ali Yilmaz; Stefanie Kliche; Ulrike Mayr-Beyrle; Guido Fellbrich; Johannes Waltenberger

Book ID
117054133
Publisher
Elsevier Science
Year
2003
Tongue
English
Weight
265 KB
Volume
306
Category
Article
ISSN
0006-291X

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p38 MAPK, but not ERK1/2, is critically
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## Abstract Combretastatin A4 (CA4) is a novel vascular disrupting agent that has promising clinical efficacy because of its ability to inhibit microtubule assembly and subsequently disrupt tumor blood flow. In this study, we demonstrate that mitogen‐activated protein kinases (MAPKs) are critically