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Oxidative damage of erythrocytes infected with Plasmodium falciparum

โœ Scribed by K. Mohan; N. K. Ganguly; M. L. Dubey; R. C. Mahajan

Publisher
Springer
Year
1992
Tongue
English
Weight
408 KB
Volume
65
Category
Article
ISSN
0939-5555

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The intraerythrocytic human malarial parasite Plasmodium falciparum produces lactate at a rate that exceeds the maximal capacity of the normal red cell membrane to transport lactate. In order to establish how the infected cell removes this excess lactate, the transport of lactate across the host cel