𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Origin and nature of correlations in the Ia feedback pathway of the muscle control system

✍ Scribed by U. Windhorst

Publisher
Springer-Verlag
Year
1978
Tongue
English
Weight
887 KB
Volume
31
Category
Article
ISSN
0340-1200

No coin nor oath required. For personal study only.

✦ Synopsis

Mammalian primary muscle spindle endings receive two inputs from fusimotor fibres and length changes of their extrafusal environment. Both inputs are also powerful noise sources disturbing the discharge patterns of the receptors. In this paper emphasis is laid on the extrafusal component. It is shown that extrafusal muscle activity can be viewed as a stochastic process which, acting as a common source, can correlate discharge patterns of adjacent muscle spindles. Some quantitative characteristics of these correlations and their relation to the underlying mechanical events are investigated in some detail in order to provide data for more theoretical considerations on their possible physiological importance.

πŸ“œ SIMILAR VOLUMES

Synthesis of optimal feedback and the st
Synthesis of optimal feedback and the stabilization of systems with a delay in the control
✍ N.V. Balashevich; R. Gabasov; F.M. Kirillova πŸ“‚ Article πŸ“… 1998 πŸ› Elsevier Science 🌐 English βš– 613 KB

The linear problem of the optimal control of systems in which the input signals contain a time delay is considered. The method of realizing optimal feedback control that is proposed is based on a special procedure for correcting the current optimal programme controls, realized by an optimal controll

Comparison of the control and pathways f
Comparison of the control and pathways for degradation of the acetylcholine receptor and average protein in cultured muscle cells
✍ Dr. Peter Libby; Dr. Alfred L. Goldberg πŸ“‚ Article πŸ“… 1981 πŸ› John Wiley and Sons 🌐 English βš– 864 KB

## Abstract In the studies reported here, we investigated whether the degradation of the acetylcholine receptor (AChR) in cultured muscle cells involves similar mechanisms as and is controlled in a manner similar to, the catabolism of the bulk of cell protein. We compared these processes after labe