Oral festination in Parkinson's disease: Biomechanical analysis and correlation with festination and freezing of gait
β Scribed by Caroline Moreau; Canan Ozsancak; Jean-Louis Blatt; Philippe Derambure; Alain Destee; Luc Defebvre
- Publisher
- John Wiley and Sons
- Year
- 2007
- Tongue
- English
- Weight
- 96 KB
- Volume
- 22
- Category
- Article
- ISSN
- 0885-3185
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## Abstract Festination and freezing of gait (FOG) are poorly understood gait disorders that cause disability and falls in people with Parkinson disease (PD). In PD, basal ganglia malfunction leads to motor set deficits (hypokinesia), while altered motor cue production leads to a sequence effect, w
## Abstract Gait festination (FE) can cause serious disability in Parkinson's disease (PD) patients. It is argued that the center of pressure position (COP) and body center of mass (COM) are possibly implicated in FE pathogenesis. The relationship between them remains unclear. The goal of this stud
## Abstract Freezing of gait (FOG) is a frequent, disabling symptom of Parkinson's disease (PD). FOG usually lasts a few seconds. It refers to brief paroxysmal events during which a subject is unable to start or continue locomotion. Despite its frequency, FOG pathophysiology is unclear. Because a f
## Abstract Background: Turning is the most important trigger for freezing of gait (FOG) in Parkinson's disease (PD), and dualβtasking has been suggested to influence FOG as well. Objective: To understand the effects of dual tasking and turning on FOG. Methods: 14 Freezers and 14 nonβfreezers match
## Abstract To revalidate the Freezing of Gait Questionnaire (FOGβQ), patients with Parkinson's disease (PD) were randomly assigned to receive rasagiline (1 mg/day) (n = 150), entacapone (200 mg with each dose of levodopa) (n = 150), or placebo (n = 154). Patients were assessed at baseline and afte