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Opposite regulation of the HPV 20-URR and HPV 27-URR promoters by ultraviolet irradiation and cytokines

✍ Scribed by Astrid Ruhland; Ethel-Michele de Villiers


Book ID
101334188
Publisher
John Wiley and Sons
Year
2001
Tongue
French
Weight
225 KB
Volume
91
Category
Article
ISSN
0020-7136

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✦ Synopsis


Epidemiological evidence implicates ultraviolet radiation and genetic changes (e.g., p53 mutations) as important factors in the etiology of nonmelanoma skin cancer. Little is known about a possible role of cutaneous papillomaviruses in these tumors. We previously reported both positive and negative regulation of the promoter activity of a number of HPV types by UV irradiation. To determine the underlying mechanism, we examined the influence of pro-inflammatory cytokines and MAP-kinases induced by UV irradiation by transfecting the HPV 20-URR and the HPV 27-URR into the RKO, HaCaT and H1299 cell lines expressing wild-type or mutated p53 or lacking p53, respectively. IL-1␣, IL-1␤, IL-6, IL-17, TNF-␣, as well as interferon-␣, -␤ and -␥ activated the promoter in the HPV 20-URR but inhibited the HPV 27-URR promoter. The effect of IL-1␣ and UV light was abolished by the addition of IL-1 receptor antagonist. UV irradiation induced a prolonged activation of JNK in HaCaT and H1299 but not in RKO cells, and its dephosphorylation was enhanced in the presence of p53 and the HPV-URRs.