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Oligodendrocytic α-synuclein accumulation causes neuronal synaptic dysfunction in a mouse model of multiple system atrophy

✍ Scribed by Ito, Hiroshi; Jin, Chenghua; Suzuki, Yasuyo; Yazawa, Ikuru

Book ID
123109170
Publisher
Elsevier Science
Year
2011
Tongue
English
Weight
112 KB
Volume
71
Category
Article
ISSN
0168-0102

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## Abstract Multiple system atrophy (MSA) is a progressive neurodegenerative disease characterized by autonomic failure, parkinsonism, cerebellar ataxia, and oligodendrocytic accumulation of alpha‐synuclein (αsyn). Oxidative stress has been linked to neuronal death in MSA and the mitochondrial toxi