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O3-1 Nocturnal oxyhemoglobin desaturation, reticulocytosis and intracranial arteriopathy in children with sickle cell disease

โœ Scribed by N. Dlamini; R.S. Bucks; S. Trompeter; M. Bynevelt; D.G. Gadian; D.E. Saunders; R. Lane; J.P. Evans; T.C. Cox; F.J. Kirkham


Book ID
114359825
Publisher
Elsevier Science
Year
2009
Tongue
English
Weight
59 KB
Volume
13
Category
Article
ISSN
1090-3798

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Mechanisms of nocturnal oxyhemoglobin de
โœ Joshua P. Needleman; Maria E. Franco; Laurie Varlotta; Darcy Reber-Brodecki; Nan ๐Ÿ“‚ Article ๐Ÿ“… 1999 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 86 KB ๐Ÿ‘ 2 views

Oxyhemoglobin desaturation in patients with sickle cell disease has been proposed as a possible mechanism in the initiaton of vasco-occlusive pain crises. Nocturnal oxyhemoglobin desaturation (NOD) has been described with a prevalence of up to 40% in children and adolescents with sickle cell disease