SUMMARY: Hypoxaemia and angiotensin II both cause pulmonary vasoconstriction and co-exist as pathophysiological pulmonary pressor stimuli in patients with cor pulmonale. Evidence from animal studies, however, suggests that angiotensin II may modulate the hypoxic pulmonary vasoconstrictor response. W
β¦ LIBER β¦
Novel Human Metabolites of the Angiotensin-II Antagonist Tasosartan and Their Pharmacological Effects.
β Scribed by Hassan M. Elokdah; Gregory S. Friedrichs; Sie-Yearl Chai; Boyd L. Harrison; John Primeau; Michael Chlenov; David L. Crandall
- Publisher
- John Wiley and Sons
- Year
- 2010
- Weight
- 29 KB
- Volume
- 33
- Category
- Article
- ISSN
- 0931-7597
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