๐”– Bobbio Scriptorium
โœฆ   LIBER   โœฆ

Nitric oxide and p53 in cancer-prone chronic inflammation and oxyradical overload disease

โœ Scribed by Julie E. Goodman; Lorne J. Hofseth; S. Perwez Hussain; Curtis C. Harris

Publisher
John Wiley and Sons
Year
2004
Tongue
English
Weight
263 KB
Volume
44
Category
Article
ISSN
0893-6692

No coin nor oath required. For personal study only.

๐Ÿ“œ SIMILAR VOLUMES

Decreased exhaled nitric oxide in sickle
Decreased exhaled nitric oxide in sickle cell disease: Relationship with chronic lung involvement
โœ Reda E. Girgis; Mohammed A. Qureshi; Judith Abrams; Paul Swerdlow ๐Ÿ“‚ Article ๐Ÿ“… 2003 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 114 KB

A deficiency in airway nitric oxide (NO) could contribute to pulmonary vaso-occlusion in sickle cell disease (SCD). We measured the fractional expired concentration of NO (FE(NO)) by chemiluminescence during a slow vital capacity maneuver against a positive pressure of 16 cm H(2)O at an expiratory f

Endothelin-1, big endothelin-1, and nitr
Endothelin-1, big endothelin-1, and nitric oxide in patients with chronic renal disease and hypertension
โœ Ivanka Mikuliฤ‡; Jรณzsef Petrik; Kreลกimir Galeลกiฤ‡; ลฝeljko Romiฤ‡; Ivana ฤŒepelak; Mo ๐Ÿ“‚ Article ๐Ÿ“… 2009 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 148 KB

## Abstract The complex pathogenesis of chronic renal disease (CRD) depends on endothelin (ET) axis (ETs and ET receptors) and nitric oxide (NO) because of their vasoactive effects and their role in general modulation of vascular homeostasis. Various renal cells synthesize ETs and NO that play a si