𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Nickel induces oxidative burst, NF-κB activation and interleukin-8 production in human neutrophils

✍ Scribed by Marisa Freitas; Ana Gomes; Graça Porto; Eduarda Fernandes

Publisher
John Wiley and Sons
Year
2010
Tongue
English
Weight
380 KB
Volume
15
Category
Article
ISSN
1432-1327

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Platelet-activating factor-induced NF-κB
Platelet-activating factor-induced NF-κB activation and IL-8 production in intestinal epithelial cells are Bcl10-dependent
✍ Alip Borthakur; Sumit Bhattacharyya; Waddah A. Alrefai; Joanne K. Tobacman; Kris 📂 Article 📅 2010 🏛 John Wiley and Sons 🌐 English ⚖ 311 KB

Background: Platelet-activating factor (PAF), a potent proinflammatory phospholipid mediator, has been implicated in inducing intestinal inflammation in diseases such as inflammatory bowel disease (IBD) and necrotizing enterocolitis (NEC). However, its mechanisms of inducing inflammatory responses a

Polyamine depletion inhibits NF-κB bindi
Polyamine depletion inhibits NF-κB binding to DNA and interleukin-8 production in human chondrocytes stimulated by tumor necrosis factor-α
✍ Annalisa Facchini; Rosa Maria BorzÍ; Kenneth B. Marcu; Claudio Stefanelli; Eleon 📂 Article 📅 2005 🏛 John Wiley and Sons 🌐 English ⚖ 265 KB

## Abstract The activation of the NF‐κB pathway by pro‐inflammatory cytokines, such as tumor necrosis factor‐α (TNFα), can be an important contributor for the re‐programming of chondrocyte gene expression, thereby making it a therapeutic target in articular diseases. To search for new approaches to

Ursodeoxycholic acid inhibits interleuki
Ursodeoxycholic acid inhibits interleukin beta 1 and deoxycholic acid-induced activation of NF-κB and AP-1 in human colon cancer cells
✍ Syed A. Shah; Yuri Volkov; Qamrul Arfin; Mohamed M. Abdel-Latif; Dermot Kelleher 📂 Article 📅 2005 🏛 John Wiley and Sons 🌐 French ⚖ 371 KB

## Abstract Deoxycholic acid (DCA) has been implicated in colorectal carcinogenesis in humans with effects on proliferation and apoptosis, mediated at least in part by activation of transcription factors nuclear factor kappa B (NF‐κB), activator protein 1 (AP‐1) and protein kinase C (PKC) enzymes.