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Neuronal Nitric Oxide Synthase (nNOS) Modulates Skeletal Muscle Hind Limb Ischemia Reperfusion Injury

✍ Scribed by S. Tsai; R. Oklu; H. Yoo; J. Martyn; W.G. Austen; M.T. Watkins; H. Albadawi


Book ID
116708641
Publisher
Elsevier Science
Year
2012
Tongue
English
Weight
43 KB
Volume
172
Category
Article
ISSN
0022-4804

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## Abstract Nitric oxide (NO), originally identified as the mediator of endothelial‐dependent relaxation of vascular smooth muscle, is now known to also have cytotoxic effects under certain conditions. Thus, we have investigated the effects of inhibition of NO synthesis on ischemia/reperfusion inju