<p>Accumulation on glia is an active pathological element in many neurological disorders. Gliosis produces neuroinflammation through both neurotrophic and inflammatory means, but the exact mechanism through which this happens remain unclear. It is suspected that damage to neurons activates the growt
Neuroinflammation in Stroke
โ Scribed by G. A. Rosenberg, S. Mun-Bryce (auth.), U. Dirnagl, B. Elger (eds.)
- Publisher
- Springer-Verlag Berlin Heidelberg
- Year
- 2004
- Tongue
- English
- Leaves
- 232
- Series
- Ernst Schering Research Foundation Workshop 47
- Edition
- 1
- Category
- Library
No coin nor oath required. For personal study only.
โฆ Table of Contents
Front Matter....Pages I-XII
Matrix Metalloproteinases in Neuroinflammation and Cerebral Ischemia....Pages 1-16
Involvement of Tight Junctions During Transendothelial Migration of Mononuclear Cells in Experimental Autoimmune Encephalomyelitis....Pages 17-38
Cytokines in Stroke....Pages 39-66
Complement Activation: Beneficial and Detrimental Effects in the CNS....Pages 67-85
Inflammation in Stroke: The Good, the Bad, and the Unknown....Pages 87-99
Tetracycline Derivatives as Anti-inflammatory Agents and Potential Agents in Stroke Treatment....Pages 101-115
Induction of Mucosal Tolerance to E-Selectin Targets Immunomodulation to Activating Vessel Segments and Prevents Ischemic and Hemorrhagic Stroke....Pages 117-132
Protective Autoimmunity and Prospects for Therapeutic Vaccination Against Self-Perpetuating Neurodegeneration....Pages 133-154
Lessons from Stroke Trials Using Anti-inflammatory Approaches That Have Failed....Pages 155-184
Inflammation-Mediated Damage as a Potential Therapeutic Target in Acute Ischemic Stroke....Pages 185-204
Design Issues in Selected Recent or Ongoing Stroke Trials....Pages 205-215
Back Matter....Pages 217-224
โฆ Subjects
Neurology
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